NURS340 PEDIATRIC NURSING

Exam 4 Study Guide

Comprehensive review covering dermatological conditions, developmental considerations, neurological disorders, and critical pediatric care concepts

04 Major Sections
30+ Clinical Topics
100% NCLEX Aligned
SECTION ONE

Dermatological Conditions

Common pediatric skin disorders, infections, and nursing management

Atopic Dermatitis (Eczema)

KEY INFORMATION

Atopic Dermatitis: Chronic inflammatory skin condition characterized by intense itching, dry skin, and eczematous lesions. Most common in infants and young children.

Clinical Manifestations

Treatment

Nursing Interventions

MRSA

KEY INFORMATION

Bacterial, highly transmissible. Contact precautions required.

Treatment

Nursing Interventions

Infection Control

Sun Exposure & Sunburn

Education

Tooth Avulsion

EMERGENCY

Tooth Avulsion: "Knocked out" tooth

Treatment

Education

Frostbite

KEY INFORMATION

Tissue damage via excessive heat loss, allows ice crystals to form in tissues

Clinical Manifestations

Treatment

SECTION TWO

Developmental Considerations

Adolescent development, hospitalized children, and growth stages

Sexual Maturation - Adolescents (Tanner Stages)

KEY INFORMATION

Puberty Assessment: Stage 1 (immature) to Stage 5 (mature)

  • Females: Breast size & shape, pubic hair
  • Males: Size & shape of penis/scrotum, pubic hair
TANNER STAGES OF SEXUAL MATURATION
Stage Females - Breast Development Females - Pubic Hair Males - Genital Development Males - Pubic Hair Approximate Age
Stage 1
(Prepubertal)
• No breast development
• Elevation of papilla (nipple) only
• Areola same color as surrounding skin
• No pubic hair
• Only vellus hair (fine body hair)
• Prepubertal
• Testes, scrotum, penis same size/proportions as early childhood
• No pubic hair
• Only vellus hair
Females: <10 years
Males: <9 years
Stage 2
(Early puberty)
Breast bud stage
• Elevation of breast and nipple as small mound
• Areola diameter enlarges
First sign of puberty in females
• Sparse growth of long, slightly pigmented hair
• Straight or slightly curled
• Mainly along labia
• Scrotum and testes enlarge
• Skin of scrotum reddens and texture changes
• Penis size usually unchanged
First sign of puberty in males
• Sparse growth of long, slightly pigmented hair
• Straight or slightly curled
• Mainly at base of penis
Females: 10-11.5 years
Males: 11-12 years
Stage 3
(Mid-puberty)
• Further enlargement of breast and areola
• No separation of contours
• Breast tissue extends beyond areola border
• Hair darker, coarser, more curled
• Spreads sparsely over mons pubis
• Penis lengthens
• Testes and scrotum continue to enlarge
• Hair considerably darker, coarser, more curled
• Spreads sparsely over pubic area
Females: 11.5-13 years
Males: 12-14 years
Stage 4
(Late puberty)
• Areola and papilla form secondary mound
• Projects above level of breast
Menarche typically occurs in Stage 4
• Hair now adult-type
• Area covered still smaller than adult
• No spread to medial thighs
• Penis increases in breadth and develops glans
• Testes and scrotum larger
• Scrotal skin darker
• Hair now adult-type
• Area covered still smaller than adult
• No spread to medial thighs
Females: 13-15 years
Males: 13-16 years
Stage 5
(Mature adult)
• Mature adult breast
• Only nipple projects
• Areola recessed to general breast contour
• Adult quantity and type
• Distribution forms inverse triangle
• Spread to medial surface of thighs
• Adult size and shape
• Genitalia mature
• Adult quantity and pattern
• Distribution forms inverse triangle
• Spread to medial surface of thighs
• May spread to linea alba in some males
Females: 15+ years
Males: 16+ years
Clinical Notes:
  • Females: Menarche (first period) typically occurs in Tanner Stage 4, approximately 2-2.5 years after breast budding begins
  • Males: First ejaculation (spermarche) typically occurs in Tanner Stage 3-4
  • Growth Spurt: Females peak in early Stage 3; Males peak in Stage 4
  • Assessment: Breast/genital development and pubic hair are assessed separately - adolescent may be at different stages for each
  • Privacy: Always assess privately and with sensitivity; explain what you're assessing and why
  • Concern: Puberty before age 8 in girls or 9 in boys = precocious puberty; No signs by age 13 in girls or 14 in boys = delayed puberty

Nursing Interventions

Routines for Hospitalized School-Age Child

CREATING STRUCTURE

Key Interventions

  • Create a daily schedule (in patient room)
  • Plan times for treatment/medications, nutrition, schoolwork, exercise, play, TV, hobbies
  • Direct communication, use of games for learning, realistic questions
  • Have what they need to cope/understand - plan with parents
  • Special toys/objects per attachment

Fears

Parent & Teen Communication

What Contributes to Poor Communication

Educate Parents

ENCOURAGE PARENTS TO
  • Accept/respect
  • Be involved
  • Avoid criticism/risky behavior
  • Promote learning
  • Clear & reasonable rules
  • Increase independence, privacy
  • Acknowledge feelings
  • LOVE!

Self-Identity: Adolescents

DEVELOPMENTAL TASK

Forming stable, coherent picture of oneself that integrates past + present experiences to gain sense for future

Components

Ego Development Stages (Ages 12-18)

ADOLESCENT DEVELOPMENT BY STAGE
Stage Physical Cognitive/Emotional Social/Identity Behavioral Characteristics
Early Adolescence
(12-14 years)
• Rapid body changes
• Growth spurts
• Clumsy coordination
• Puberty begins/progresses
• Concrete thinking predominates
• Beginning abstract thought
• Increased day-dreaming
Lacks impulse control
• Mood swings
Conformity to peer group
• Same-sex friendships primary
• Defining independence from family
• Preoccupied with body changes
• Self-conscious about appearance
• Testing boundaries
• Seeking peer approval
• Comparing self to others
• Experimentation begins
• Privacy increasingly important
Middle Adolescence
(15-16 years)
• Physical maturation continues
• Most rapid growth completed
• Sexual characteristics developing
• Body image concerns peak
• Abstract thinking developing
• "Imaginary audience" thinking
Feelings of omnipotence & invincibility
• Risk-taking mentality
• "It won't happen to me"
• Defining sexual identity & orientation
• Intense peer relationships
• May have romantic relationships
Concerned with appearance
• Complaints about interfering parents
• Maximum parent-teen conflict
• Peak risk-taking behaviors
• Strong need for independence
• Experimentation increases
• May challenge authority
• Peer influence strongest
Late Adolescence
(17-18 years)
• Physical maturation nearly complete
• Adult body proportions
• Coordination improved
• Less preoccupied with body
Firmer identity
• Able to delay gratification
• Able to think ideas through
• Capable of useful insight
• More developed sense of humor
• Able to express ideas in words
• Stable interests developing
Moral/ethical/spiritual values develop
• Mutual relationships with opposite sex
• Giving/sharing in stable relationships
• External romance
• More comfortable with self
Emotional stability
• Caring & compassionate
• Pride in work
• Caring for self
• Constancy of emotion
• Planning for future
• More realistic thinking
Erikson's Stage: All of adolescence (12-18 years) = Identity vs. Role Confusion
  • Task: Develop coherent sense of self and role in society
  • Success: Strong sense of identity, fidelity to beliefs and people
  • Failure: Role confusion, inability to commit, identity diffusion
  • Key Question: "Who am I and where am I going?"
QUICK REFERENCE

High-Yield Facts for Exam

CRITICAL "NEVER" STATEMENTS
  • NEVER give children aspirin (Reye's syndrome risk)
  • NEVER give infants honey before age 1 (botulism risk)
  • NEVER use children/family as translators (use professional interpreters)
  • NEVER tell siblings "they went away" or "they're sleeping" (use clear language)
CRITICAL TIME FRAMES
24 hours Isolation for suspected meningitis (minimum)
24-48 hours Monitoring after near drowning (delayed complications)
30-35 minutes Status epilepticus = medical emergency
1 hour Tooth avulsion - must reimplant within 1 hour
2 years Seizure-free before discontinuing meds (+ normal EEG)
80-85% Spina bifida patients develop hydrocephalus
OPPOSITES TO REMEMBER
  • TETANUS = RIGID (muscles stiff, locked, spastic, patient alert)
  • BOTULISM = FLOPPY (hypotonic, weak, decreased reflexes)
  • Decerebrate → Decorticate = IMPROVEMENT (Reye's syndrome)
  • Bacterial meningitis = HIGH fever, very ill, bacteria in CSF
  • Viral meningitis = Moderate fever, less ill, NO bacteria in CSF
TESTABLE ASSESSMENTS

Glasgow Coma Scale

  • Score of 15 = BEST
  • Three categories: Eye opening, Motor response, Verbal response
  • Pediatric version for children under 2

Nuchal Rigidity

  • Hallmark sign of meningitis (older children)
  • Qualitative assessment - cannot measure
  • Difficulty/inability to flex neck forward

Bulging Fontanelles

  • Sign of increased ICP in infants
  • Assess when infant is calm (crying = normal bulging)
SHUNT MALFUNCTION = EMERGENCY

Signs parents MUST know:

  • Fever
  • Inflammation along shunt tract
  • Abdominal pain
  • Vomiting
  • Changes in LOC
MEDICATION HIGHLIGHTS
Phenobarbital Give with Vit D + folic acid, can give with milk
Phenytoin (Dilantin) Give with Vit D + folic acid, DO NOT give with milk
Diastat Rectal diazepam - first line for status epilepticus
Versed Intranasal midazolam - second line status epilepticus
Ativan IV lorazepam - third line status epilepticus (emergency)
Accutane HIGHLY TERATOGENIC - effective contraceptives required
SPINA BIFIDA CLASSIFICATION
Type Definition Clinical Presentation Neurological Impact Treatment Prognosis
Spina Bifida Occulta
("Hidden")
• Mildest form
• Failure of vertebral arch fusion
Meninges NOT involved
• Spinal cord intact and normal
• Often undiagnosed
SKIN INDICATORS:
• Sacral dimple
• Dark tufts of hair
• Port wine nevus/angiomas
• Lipomas in sacral area

Usually asymptomatic
• Usually NONE
• Normal neurological function
• No paralysis
• Bowel/bladder control normal
• May have tethered cord in some cases
• Usually no treatment needed
• Monitor for signs of tethered cord
• May need surgery if tethering occurs
• Parent education about what to watch for
Excellent
• Normal life expectancy
• Normal activities
• Most people never know they have it
Meningocele • Moderate form
• Meninges (sac) protrudes through vertebral defect
Does NOT contain neural tissue
• Spinal cord and nerves normal
• Visible sac on back
• Filled with CSF
• Covered with meninges and skin
• Can occur anywhere along spine
• Most common in lumbar-sacral area
Usually minimal neurological deficits
• May have minor weakness
• Bowel/bladder function usually intact
• Risk of hydrocephalus lower than myelomeningocele
Surgical repair to close defect
• Usually done in first few days of life
• Protect sac pre-op (moist, sterile)
• Prone positioning pre-op
• Monitor for infection
Good to Excellent
• Near-normal function
• May have minor limitations
• Good quality of life
Myelomeningocele
(Most severe)
• Most severe form
Both meninges AND spinal cord/nerve roots protrude
• Neural tissue exposed or covered by thin membrane
• Usually accompanied by Chiari II malformation
VISIBLE SAC containing neural tissue
• May leak CSF
• High infection risk
• Most common site: lumbosacral
• Level of defect determines impairment severity
SIGNIFICANT neurological deficits:
• Paralysis below defect level
• Loss of sensation
• Neurogenic bowel/bladder
80-85% develop hydrocephalus
• May have Arnold-Chiari malformation
• Orthopedic deformities common
PRE-OP:
• PROTECT SAC (moist, sterile)
• Prone position
• Prevent infection
• Monitor head circumference

SURGERY:
• Close defect within 24-48 hours

POST-OP:
• Watch for increased ICP
• May need VP shunt
• Lifelong management
Variable
• Depends on lesion level
• Requires lifelong care
• Multiple surgeries common
• Mobility aids often needed
• CAN have good quality of life with support
• Many complications possible
CRITICAL POINTS:
  • 80-85% of myelomeningocele patients develop hydrocephalus - often requires VP shunt placement
  • Latex allergy: HIGH RISK in ALL types - use latex-free equipment from birth
  • Level of lesion = level of function: Higher lesions = more impairment
  • Folic acid prevention: 400 mcg daily BEFORE pregnancy reduces risk by 70%
  • Pre-op priority: PROTECT THE SAC - keep moist, sterile, prone position
SKIN INDICATORS OF SPINA BIFIDA OCCULTA (Know These)
  • Sacral dimple
  • Port wine nevus or angiomas
  • Dark tufts of hair in sacral area
  • Lipomas in sacral area
PRE/POST-OP MYELOMENINGOCELE

Pre-Op

  • PROTECT THE SAC - warm, moist, sterile saline
  • PRONE POSITION
  • Warming bed
  • Frequent stool cleaning/hourly diapers
  • Head circumferences

Post-Op

  • NO MORE SAC! Now surgical incision
  • Keep prone or side-lying for feeds
  • Watch for increased ICP (contents now internal)
  • Monitor for SIDS risk (prone positioning)
  • Can breastfeed (position carefully)
  • Continue head circumferences
LATEX ALLERGY IN SPINA BIFIDA

High risk due to repeated exposures

Foods that cross-react: Banana, avocado, kiwi, chestnuts

Use latex-free equipment for ALL procedures

END OF STUDY GUIDE

Good Luck on Your Exam!

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